A study published in 2011 conducted on 1,400 randomly selected residents from Queensland aged 20-54 years, reported that 83% of the participants had premature skin ageing, worsening after the age of 30. The conclusion of this study was that premature skin ageing is common. It is associated with sun exposure during leisure or work, and therefore is preventable.1
Ultraviolet radiation from the sun has deleterious long-term effects on human skin, leading to premature ageing (photoageing). One of the most obvious acute effects of UV on the skin is the generation reactive oxygen species (ROS) and the induction of inflammation. In the epidermis, UV exposure has dramatic effects on cell cohesion and mechanical integrity of the stratum corneum, compromising the critical barrier function of the skin. Dermal damage induced by ultraviolet radiation is principally manifested histologically as the disorganisation of collagen fibrils. Ultraviolet radiation can induce several types of epidermal injury, including sunburn cell formation.
Selection of biological markers
In order to follow the UV-induced damage, some biological targets were identified as markers of the photoageing condition (Fig 1), and were used to assess the antiphotoageing effect of Cherry Blossom Eutectys™ BG.
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